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Submitted: 29 Oct 2021
Revision: 16 Apr 2022
Accepted: 29 Apr 2022
ePublished: 14 Jun 2022
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J Cardiovasc Thorac Res. 2022;14(2): 90-94.
doi: 10.34172/jcvtr.2022.15
PMID: 35935383
PMCID: PMC9339735
Scopus ID: 85140248694
  Abstract View: 723
  PDF Download: 487
  Full Text View: 97

Original Article

The relationship between radial artery spasm and adropin levels in patients undergoing transradial coronary angiography

Fuat Bice 1 ORCID logo, Mehmet Eyuboglu 1* ORCID logo, Zeliha Cansel Ozmen 2, Baris Acikel 1, Mustafa Yilmaz 1, Metin Karayakali 1, Kayihan Karaman 1, Cagri Zorlu 1, Atac Celik 1

1 Department of Cardiology, Gaziosmanpasa University School of Medicine, Tokat, Turkey
2 Department of Biochemistry, Gaziosmanpasa University School of Medicine, Tokat, Turkey
*Corresponding Author: Corresponding Author: Mehmet Eyuboglu, Email: , Email: mhmtybgl@gmail.com

Abstract

Introduction: Transradial coronary angiography (TRA) is associated with a lower incidence of bleeding rate and access site complications and is associated with better outcomes compared to transfemoral angiography. However, radial artery spasm (RAS) is an important limitation of TRA procedures. Little is known regarding the relationship of serum vasodilator and inflammatory markers with RAS. Therefore, the present study aimed to investigate the association between serum adropin level and RAS in patients undergoing TRA.

Methods: From February 2020 to January 2021, 39 consecutive patients who underwent elective daiagnostic TRA and experienced RAS during the procedure, and 42 age and sex matched controls who did not experience RAS were prospectively included into the study. The groups were compared regarding serum adropin levels and inflammatory markers.

Results: Although adropin levels were found to be lower in the RAS group, this difference was not statistically significant between the the patients with RAS and controls (14.9 vs. 16.1, P=0.105). However, inflammatory parameters monocyte count and MHR (monocyte/HDL cholesterol ratio) were found to be statistically significantly higher in the RAS group compared to controls (P=0.001 and P=0.010, respectively). Moreover, a significant positive correlation was found between the monocyte count and RAS (r:0.360, P<0.001), and between MHR and RAS (r:0.288, P=0.009). Furthermore, multivariate analysis demonstrated that monocyte count (OR:1.671, 95%CI:1.312-2.094, P=0.001) and MHR (OR:1.116, 95%CI:1.054-1.448, P=0.022) were found to be independent predictors of RAS.

Conclusion: Serum vasodilator and inflammatory markers may be useful in the prediction of RAS in patients undergoing TRA procedures.

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